THE PRACTICE TEST
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- Questions
- 33
- Format
- Multiple choice
- Access
- 2 years
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- Explanations
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Sample practice questions
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is classified as a mast cell release inhibitor is one of the substances released during an allergic response is found in white blood cells known as neutrophils does not cause skin reactions
are only found in the bloodstream are coated with immunoglobulins that recognize antigens have no other role than antiallergy in the body degranulate when the H 1 -receptor is stimulated
can be blocked only by histamine cannot be prophylactically treated is characterized by itching, sneezing, and edema is only characterized by wheals and urticaria
are the newest, third-generation drugs are effective and can cause drowsiness are not found in OTC products do not have anticholinergic properties
are more selective for peripheral H 1-receptors bind the H 1 -receptors in the brain to produce fewer side effects are more sedating and drying than diphenhydramine are mast cell stabilizers
histamine after it is released H 1 -receptors from binding histamine the release of histamine and other active substances from mast cells the effect of histamine after it binds
promethazine cetirizine loratadine levocetirizine
epinephrine (Adrenaline) ipratropium (Atrovent) salmeterol (Serevent) theophylline (Elixophylline)
fluticasone cromolyn albuterol montelukast
anticholinergics beta adrenergics methylxanthines corticosteroids
produce bronchodilation increase expectoration of mucus inhibit release of mast cell mediators liquefy bronchial secretions
cromolyn omalizumab ipratropium guaifenesin
ECF-A leukotrienes only prostaglandins and leukotrienes all of these
bronchodilation increased cyclic AMP increased mucus secretion stimulation of the beta-2 receptors
inhibit release of mediators from mast cells block beta-2 receptors activate beta-receptor and relax smooth muscle only inhibit release of mediators from mast cells and relax smooth muscle
decrease activation of arachidonic acid decrease the expression of pro-inflammatory mediators and production of pro-allergic antibodies increase the production of pro-allergic antibodies only decrease activation of arachidonic acid and decrease the production of allergic antibodies
piperacillin vancomycin gentamicin clindamycin
erythromycin tetracycline gentamicin clindamycin
lipid soluble well absorbed orally poorly absorbed from the GI tract and excreted mostly unchanged in the urine if injected devoid of serious adverse effects
binds to keratin is fungicidal forms ergosterol in hair and nails incorporates itself into fungal RNA
systemic infection may occur in patients who are receiving drugs to suppress their immune system warts are an example of a dermatophytic fungal infection the majority of antifungal drugs interfere with fungal cell-wall integrity amphotericin B is associated with serious renal toxicity
echinocandins inhibit polysaccharide (glucan) synthesis in the cell wall flucytosine binds to the cell membrane ticonazole is an antimetabolite for fungal RNA amphotericin B binds to a cytochrome P450-dependent enzyme
butenafine gentian violet clotrimazole enfuvirtide
changing cell-wall synthesis binding to ergosterol inhibiting cell mitosis inhibiting neuraminidase
immunity can be gained through the attenuated live virus treatment can be given via an intranasal spray the vaccines are contraindicated in people allergic to eggs effective treatment occurs only with respiratory drug administration
mercaptopurine fluorouracil methotrexate cytarabine
doxorubicin asparaginase etoposide vincristine
tamoxifen anastrozole leuprolide goserelin
imatinib trastuzumab bevacizumab gefitinib
one of the early goals of chemotherapy is remission antineoplastic drugs only attack cancerous cells drug resistance is increased with combination therapy it is the primary form of treatment for solid tumors
decrease cells' ability to synthesize proteins are cell-cycle nonspecific bind irreversibly with nucleic acids and are cell-cycle nonspecific all statements are true
blocking DNA synthesis forming abnormal nucleotides preventing DNA replication and forming abnormal nucleotides all of these
they can bind to microtubules they induce enzymes they increase DNA strand breakage they can bind to microtubules, and they increase DNA strand breakage
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